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For over 40 years, Biotics Research Corporation has revolutionized the nutritional supplement industry by utilizing “The Best of Science and Nature”. Combining nature’s principles with scientific ingenuity, our products magnify the nutritional
eStoreRx™ is an easy direct-to-patient ordering & fulfilment program for lifelong wellness.
Biotics Research is proud to expand our commitment to education with the Wellness Unfiltered Pro Podcast. Each episode delves into key health topics and the clinical applications of our premier products. Through candid, insightful conversations, our team offers practical guidance to keep you informed and empowered as a healthcare professional.
January 23 2025
Environmental Science and Technology Letters recently published an analysis of the per- and polyfluoroalkyl substances (PFAS) content of 22 different ...
This month an analysis of National Health and Nutrition Examination Survey (NHANES) data, which included over 40,000 participants was published, indicating an association between exposure to the insecticide malathion and impaired kidney function. Malathion is the most widely used organophosphate insecticide in the US, used in thousands of products, as well as for agricultural purposes and mosquito control. It is worth noting that 15% of the US adult population has chronic renal disease, and as many as 90% do not know it. In this study, malathion exposure (determined by urinary metabolite) was associated with a 26% higher risk for low kidney function, and the association was found even after excluding participants with either diabetes or hypertension. It’s also worth noting that cadmium levels had a similar risk for impaired kidney function; because this association is well-established, it was used as a positive control.
Although a cross-sectional study and limited by the nature of the testing (urine pesticide and kidney function testing was done only once), this is not the first study to suggest a link between pesticides and kidney damage. Commercial pesticide applicators have a higher risk for end-stage renal disease as well as a reduced eGFR, and acute malathion exposure has been linked to acute kidney damage in previously published case studies. While the NHANES study did not provide a mechanism, malathion toxicity appears to be mediated, at least in part, through oxidative stress, DNA damage, and mitochondrial dysfunction, and suggests mitigation through glutathione support (e.g., N-acetylcysteine, lipoid acid, vitamin C, etc.) to be a reasonable strategy.
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